trpv4 pain
Our results suggest that microglial TRPV4 channels reside at the center of the neuroimmune axis in the spinal cord, which transforms peripheral nerve injury into central sensitization and neuropathic pain, thereby identifying TRPV4 as a potential new targ,由 VL Ehlers 著作 · 2023 · 被引用 5 次 — Peripheral transient receptor potential vanilloid type 4 hypersensitivity contributes to chronic sickle cell disease pain · Authors · Affiliation. ,由 P Kanju 著作 · 2016 · 被引用 74 次 — Our novel compounds attenuate pain behavior in a trigeminal irritant pain model that is known to rely on TRPV4 and TRPA1. ,由 CTD Antoniazzi 著作 · 2024 — This review summarizes the role of TRPV4 in cancer etiology and cancer-induced pain mechanisms, as well as the current status of the clinical research. ,由 S Fozzato 著作 · 2021 · 被引用 8 次 — TRPV4 and TRPM8 channels may play a role in CLBP and warrant further investigations as possible therapeutic targets. ,由 N Alessandri-Haber 著作 · 2005 · 被引用 257 次 — The ligand-gated ion channel, TRPV4, functions as a transducer of hypotonic stimuli in primary afferent nociceptive neurons and contributes to inflammatory ... ,由 P Rodrigues 著作 · 2023 · 被引用 13 次 — The TRPV4 gene mutations have generated osteoarthropathy, skeletal dysplasia and peripheral neuropathies, manifesting as a variable combination ... ,由 L Michalick 著作 · 2020 · 被引用 101 次 — Emerging evidence has implicated TRPV4 in the activation and differentiation of innate immune cells, especially in neutrophils, monocytes, and macrophages. ,由 Z Qin 著作 · 2023 · 被引用 3 次 — In summary, MJS significantly inhibited pain and itching behavior, as well as itching may be caused by TRPV4 activation. For the first time, an inhibitory ...
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 trpv4 pain 相關參考資料
A TRPV4-dependent neuroimmune axis in the spinal cord ...
Our results suggest that microglial TRPV4 channels reside at the center of the neuroimmune axis in the spinal cord, which transforms peripheral nerve injury into central sensitization and neuropathic ... https://www.jci.org Peripheral transient receptor potential vanilloid type 4 ...
由 VL Ehlers 著作 · 2023 · 被引用 5 次 — Peripheral transient receptor potential vanilloid type 4 hypersensitivity contributes to chronic sickle cell disease pain · Authors · Affiliation. https://pubmed.ncbi.nlm.nih.go Small molecule dual-inhibitors of TRPV4 and TRPA1 for ...
由 P Kanju 著作 · 2016 · 被引用 74 次 — Our novel compounds attenuate pain behavior in a trigeminal irritant pain model that is known to rely on TRPV4 and TRPA1. https://www.nature.com Targeting TRPV4 Channels for Cancer Pain Relief
由 CTD Antoniazzi 著作 · 2024 — This review summarizes the role of TRPV4 in cancer etiology and cancer-induced pain mechanisms, as well as the current status of the clinical research. https://www.mdpi.com TRPV4 and TRPM8 as putative targets for chronic low back ...
由 S Fozzato 著作 · 2021 · 被引用 8 次 — TRPV4 and TRPM8 channels may play a role in CLBP and warrant further investigations as possible therapeutic targets. https://link.springer.com TRPV4 mediates pain-related behavior induced by mild ...
由 N Alessandri-Haber 著作 · 2005 · 被引用 257 次 — The ligand-gated ion channel, TRPV4, functions as a transducer of hypotonic stimuli in primary afferent nociceptive neurons and contributes to inflam... https://www.sciencedirect.com TRPV4 Role in Neuropathic Pain Mechanisms in Rodents
由 P Rodrigues 著作 · 2023 · 被引用 13 次 — The TRPV4 gene mutations have generated osteoarthropathy, skeletal dysplasia and peripheral neuropathies, manifesting as a variable combination ... https://www.ncbi.nlm.nih.gov TRPV4—A Missing Link Between Mechanosensation and ...
由 L Michalick 著作 · 2020 · 被引用 101 次 — Emerging evidence has implicated TRPV4 in the activation and differentiation of innate immune cells, especially in neutrophils, monocytes, and macrophages. https://www.frontiersin.org Vitexin inhibits pain and itch behavior via modulating ...
由 Z Qin 著作 · 2023 · 被引用 3 次 — In summary, MJS significantly inhibited pain and itching behavior, as well as itching may be caused by TRPV4 activation. For the first time, an inhibitory ... https://www.sciencedirect.com |